neo-x wrote:This stage may start with singular cellular life, but then the gap is how do we get the information rich biological code and it's arrangement thereof, specific functions of RNA and proteins alongside DNA
I think this right here is what is the crux of your argument. If I may, I'd say that I look at it as not as an increase in information. It's just a different arrangement of the same building blocks, nothing has been changed or added to them. To think that life at a microscopic level is less complex, is not the same as to say, therefore easier to have had formed.
And another problematic phrase you use is
"and mutations acting upon such genetic information and biology". and then
"for increasing the genetic information so much"
Mutations don't act on genetic information. There is no such thing. A mutation is not functional - preset code which comes and fights with the DNA code to overcome it. When the DNA, or "information" as you call it - the building blocks of life, gets garbled because of radiation or a replicating problem occurs where the cell can't part with the right ingredients or misses a chromosome or two in the process or miss copying a gene, is what is called a mutation. The mutation doesn't arrive from somewhere. And we know that happens today still, the DNA molecule doesn't replicate perfectly there are always problems. The only kicker is that if the said garbled arrangement of the molecule works in favor of its host in the environment it lives in or not; and could the host pass it on successfully. If the host survives, it copies the garbled genes on and thus you say it is increased information. But it's not increased info in the same sense that it's (externally) added information. Its just the same DNA molecule which is partly arranged in a way it was not before. If the garbled information or mutation as you say, doesn't help the host even if the host passes it on, it will go down in the gene pool as a minor variation and less likely to occur or may go completely away.
Edit:
To take an example...let's say for the sake of simplicity the original sequence in the DNA was:
AGGT CTGG CGAT TTGA AACC TGCA GAAC...
and via UV radiation of the sun the molecule rearranged itself to
ACGT CCGG AGAT TCGA CACT TGCA GAAC.
Compare the two and you see slight variations. Now how this may result in the organism? It may be that it formed a part of a visual sensor the or hearing sensor or skin change or maybe it formed a nose. Whatever function you think of, the variation in the garbled code could make the particular function inert, malfunctions it, or forms it in a slightly different way. That's mutation, and if this change helps the host, then good, if not then the change may cause the host to die and the host eventually can go extinct. Just one of the many possibilities, e.g overlapping of sequence, clipping of it, etc. can all result in different changes.
Yes, this is what I meant by "mutations acting upon such genetic information and biology", although if I were more careful I'd reword this to something more like "mutations happening in genetic information." It's not so much that "mutations" act (like say Natural Selection acts), but more meant the genetic information is needed for mutations to occur. Otherwise I see nothing wrong with you you've written; but it does accentuate my point that this "critical mass" (which I'll try explain below) is needed in order for the main evolutionary mechanisms (NS acting upon mutations) to do their thing.
Neo-X wrote:If you saw the video (I detest his quibs on creationists) you see the same thing explained.
By the way, the same happens in humans, rats and chimps and even reptiles. Humans carry genes for producing egg yolk, full set of fur coat, male nipples etc. Chimps and rats carry the same genes of the fur coat but in their development the gene is functional, it hasn't been garbled, in humans, these genes are broken the sequence is there but with slightly rearranged, mutated - garbled code, its there but can't fully function.
This is why when people say God made everything with the same material is good on a superficial level because we tend to think that the code doesn't exist in us for things that don't work in us. Like we are made exactly for the things we do today. But it does, we still carry egg-yolk making sequence when our ancestors delinked from reptiles. It's still there just slightly broken.
I'd like to respond to this, and really, when we're talking psudeogenes, I find biologists start thinking themselves theologians. They raise theological arguments to do with God and God's design rather than focus on evolution. Trying to prove evolution through falsifying what they see as an outrageous competing position (aka certain creation positions). Falsification of another position, doesn't prove your own position (someone should tell that to Jerry Coyne
).
There is an often made formal fallacy by many evolutionary scientists too, that is, affirming the consequent. Note, the following logic doesn't follow:
- 1) If P then Q
2) Q is true
3) Therefore P
It's easy to do, I'm sure I've done it, we all do it. So then, let me substitute in the argument from the egg-yolk gene found in humans that is apparently non-functional.
- 1) If 'we are descended from reptiles' then 'we should carry non-functional genes'.
2) We do carry non-functional genes (Vitellogenin "egg-yolk" genes)
3) Therefore, these egg-yolk making sequences we carry are left over from when our ancestors delinked from reptiles
Note, the conclusion doesn't follow from the argument presented. Yes, you do have a confirmed prediction, and one that might be quite troubling for those who believe God created life forms. It is good evidence, but it is not equivalent to proving that we are descended from reptiles.
Clearly evolutionary theory is consistent with vitellogenins. The same is true for other patterns we observe also. Such patterns can fall into an evolutionary common descent pattern, or they may not. One thing I find troubling with evolutionary theory as scientists use it, is that if a sequence is not the case, then evolution can account for that too. Evolution is used to explain a wide range of observable features, that's great right? The downside is that it becomes less falsifiable, and in doing so, evidential arguments are less forceful. If it can explain A, and B, and C, then when we find 'C' it isn't compelling evidence for the theory.
Let me use the egg yolk example, referencing Dennis Venema's BioLogos article:
Vitellogenin and Common Ancestry: Understanding synteny:
- Modern birds (such as chickens) have three vitellogenin genes: VIT1, VIT2, and VIT3. The latter ones sit side by side in the chicken genome, with VIT1 in a different location. The three VIT genes sit next to other genes in the chicken genome: VIT1 sits next to a gene called “ELTD1”, and VIT2 and VIT3 sit between genes named “SSX2IP” and “CTBS”. These genes are not involved in making egg yolk - they just happen to be the closest neighbors of the VIT genes.
...
researchers f[ou]nd fragmentary remains of all three VIT genes in the human genome (though the human VIT1 sequence was the best preserved of the three) they also found sequence matches that span both regions on either side of the genes in question. This evidence increases our confidence that we are indeed looking at regions with shared synteny: in other words, a region in two present-day species that was once a region in the genome of their common ancestral population. The human VIT sequences, as expected, are far too fragmentary to act as functional VIT genes.
So then, from this, the vitellogenin genes in chickens share a weak similarity with corresponding genetic segments in humans. Venema (and many evolutionary scientists) view the human segments as pseudogenes (non-functional vitellogenin genes inherited from their egg-laying ancestors). The conclusion, vitellogenin similarity between humans and chickens proves humans and chickens evolved via mutations from a common ancestor (
note the affirming the consequent fallacy).
Yet, despite the conclusion not logically following, it is also argued that similarity of the vitellogenin genes between humans and chickens is further confirmed by their positioning within their respective genomes (synteny). This is something common descent through evolution would predict, right? (C) But then, if there was loss of synteny (B) with the positioning was found elsewhere, even dramatically so (A) evolution still wouldn't be harmed. The theory has a range of explanatory mechanisms every outcome that might be found (A, B and C). This means these apparent evidentiary arguments don't count as very forceful evidence, and indeed it seems impossible to falsify full-blown common descent via evolution which I why I don't even bother to try. I'd prefer not to
and just let people believe saturated with such thought believe it, while knowing to my own rational satisfaction reasons why I believe in my form of progressive creation.
Of course, things like the yolk sac, and egg-yolk gene, this particular apparently non-functional psuedogene, when I bring such information back to my theological beliefs, and beliefs on creation, I need to work through it in order for my beliefs to be consistent with reality. Any rationalist, which I consider myself as being, ought to and, that I believe I do best as I can. It is part of loving God with my mind as far as I see matters.
Neo-x wrote:Take cross-breeding which can occur. Cross-breeding couldn't occur in the beginning, until a critical mass is hit where life has taken hold that allows cross-breeding to occur.
I am not sure what this means, k. You don't need cross breeding to occur, you only need the DNA to branch out. What do you mean by critical mass?
How does DNA branch out? One way I can logically conceive, is via cross-breeding. Another is genetic transposition. Then mutations (like you pointed out happens with increased radiation), and NS acting on genetic code. But, how does the initial genetic code get there which can then possibly be shared across species, duplicated and modified?
A "critical mass" of such genomic information is required. A critical mass of "life", and I'd say "life forms", is required before natural selection can work it's magic. Otherwise there can be no mutations. First, an accumulation of life is necessary, an accumulation of genetic information within populations (and by all accounts being qualified in Information Technology, I think I'm fit to see that such is clearly "information" on different levels), then once this is had speciation can take place via natural selection acting upon mutations. But prior to this "critical mass" being had (i.e., minimum size or amount of life and biological information required to start Darwinian forms of evolution), other mechanisms are required to offer up a complete picture that all life is in fact descended from single cellular life such as say, unicellular algae.
Neo-x wrote:Link added:
Also, read this small link about the evolution of cecal valves in Podarcis sicula, a species of lizard. Ofcourse you can find more material on it. But my point with this is, how new function or information, as you put it, forms through evolution, on its own. Cecal valves only appear in 1% in scaled reptiles. This species didn't have them but in a couple fo decades they did.
https://www.sciencedaily.com/releases/2 ... 112433.htm
Thanks, I did find it interesting. From the article, I read that the genome of the adapted lizards on the new island, is still the same i.e., "
Tail clips taken for DNA analysis confirmed that the Pod Mrcaru lizards were genetically identical to the source population on Pod Kopiste." This to me points to a mechanism I referred to in one of my earlier posts as a biological plasticity.
Yes, we can adapt, some species more than others, and this results in morphological changes -- yet, the plasticity is accounted for in the biology of the species. Kind of like an elastic band. It is similar to finches beaks, during drought the seeds eaten by the finches became tougher, and birds with bigger beaks able to survive and reproduce. After the drought ended, seeds returned beaks also returned back to pre-drought sizes.
In your profile signature, you have, "
The thing is I find it funny when people accept micro evolution but not macro its like saying I believe in inches but not miles." This is one reason why some mechanisms (for better or wrose, often boxed into "microevolution") do not allow for miles. A finch remains a finch, the Italian lizard as it lizard, despite these adapative changes. It's like they have an elastic band on them, which allows them to travel inches and not miles. Unless, there is a way to snap that rubber band and re-encode, if you will, their genome (which require other mechanisms to be brought to bare than I think those displayed with finches and these lizards).