Posted: Tue Nov 08, 2005 10:26 pm
gone
"The heavens declare the glory of God; the skies proclaim the work of his hands." (Psalm 19:1)
https://discussions.godandscience.org/
I fail to see what your issue is, and do not see that the study of coral layers makes an error?Jbuza wrote:I think you should file the study you quoted me in the garbage pail as the propaganda that it is. It is based on presuppositions, and is offered as proof of that presupposition. Length of days would have minimal impact on growth rates when compared to factors like quantity and type of minerals dissolved in the oceans at any particular time, the amount of cloud cover on any particular day, the temperature of the ocean at any particular time. The study isn't even remotely convincing of anything except the large bias of the researcher that is evident in his coming to nonsensical conclusions to support his presupposition. Rate of coral growth as I said before is another good example of the idiocy of uniformitarianism. Growth rate of coral is far more variable than the length of days has ever been.
Stability in natural laws is "Evolution"? How on earth does one ever draw that conclusion. Is gravity being uniform throughout time evolution? I just don't get it.Jbuza wrote:Uniformitarianism is Evolution.
While evolution is to be taught in state schools, we believe it is wise for teachers to encourage their pupils to examine Darwinism critically. Henderson, while claiming to endorse “critical thinking,” actually shows what he really thinks about the scientific method when he stated that such a critical attitude to evolution “must be resisted.” My seventeen years of teaching science in state comprehensive schools3 were motivated by encouraging children to think scientifically and critically. The uncritical acceptance of unproven (indeed, unprovable) evolution is contrary to scientific methodology and good science teaching.
Here's another quote from this article.IRQ Conflict wrote:Here is some more on the matter regarding evolutionary thought in regards to medicine.While evolution is to be taught in state schools, we believe it is wise for teachers to encourage their pupils to examine Darwinism critically. Henderson, while claiming to endorse “critical thinking,” actually shows what he really thinks about the scientific method when he stated that such a critical attitude to evolution “must be resisted.” My seventeen years of teaching science in state comprehensive schools3 were motivated by encouraging children to think scientifically and critically. The uncritical acceptance of unproven (indeed, unprovable) evolution is contrary to scientific methodology and good science teaching.
How does that work? Destroying information leads to resistance?The antibiotic resistance of the MRSA bacteria (methicillin resistant Staphylococcus aureus, sometimes known as the “Superbug”) is due to mutations destroying the information that allows it to resist antibiotics. There is no increase in information.
BGoodForGoodSake wrote:Here is another link on Uniformitarianism and how one might confuse its actual meaning without a proper historical context.
Evolution ignores that which it cannot explain, because it isn't interested in sincere discovery, and is only committed to itself and the explanations of life without God. Morality and the goodness of man comes from the Spirit of God and this world is hurrying down a path toward acceptance of behavior and rejection of constraint. This journey is more important than discovery of TRUTH.
BGoodForGoodSake wrote:How does that work? Destroying information leads to resistance?
Another mechanism of resistance is what occurs when a mutation takes place that might, for example, cause a defect in the bacteria's ability to transport the antibiotic into the cell, thus rendering the bacteria resistant to that particular antibiotic. Another mutation might change a binding site used by the antibiotic within the cell, thus rendering it unable to kill the cell. What is never brought up, however, is the fact that any mutation will result in a loss of information due to the change in genetic material. Even in the very unusual occurrence of a so-called “beneficial” mutation, there is an ultimate loss of genetic information available to succeeding generations.
linkRobert T. (Tommy) Mitchell, M.D., is a graduate of Vanderbilt University School of Medicine and practices Internal Medicine in Gallatin, Tennessee. He is Board Certified in Internal Medicine and is a Fellow of the American College of Physicians.
linkFor example, bacteria may become resistant to to macrolides (erythromycin, azithromycin, clarithromycin, dirithromycin, troleandomycin, etc.) by producing a slightly altered 50S ribosomal subunit that still functions but to which the antibiotic can no longer bind (see Fig. 3A). Bacteria may become resistant to beta-lactam antibiotics (penicillins, monobactams, carbapenems, and cephalosporins) by producing altered transpeptidases (penicillin-binding proteins) with greatly reduced affinity for the binding of beta-lactam antibiotics. Bacteria may become resistant to vancomycin by producing altered cross-linking peptides in the peptidoglycan to which the antibiotic no longer bonds. Bacteria may become resistant to fluoroquinolones (norfloxacin, lomefloxacin, fleroxacin, ciprofloxacin, enoxacin, trovafloxacin, etc.) by producing altered DNA gyrase or topoisomerases
No, the list is a list of misconceptions. Unifomitarianism is not unique to geology and is not constant across time without catastrophic events.IRQ Conflict wrote:
Thanks Bgood! I didn't know that "Uniformitarianism is unique to geology" So it appears that uniformitarianism holds geologic rates that were constant across time without any catastrophic events. Is that a good description?
This statement is misleading, your second source cleary shows this is not the case. Loss of information here is from the perspective of the antibiotic, but why take this perspective?IRQ Conflict wrote:Another mechanism of resistance is what occurs when a mutation takes place that might, for example, cause a defect in the bacteria's ability to transport the antibiotic into the cell, thus rendering the bacteria resistant to that particular antibiotic. Another mutation might change a binding site used by the antibiotic within the cell, thus rendering it unable to kill the cell. What is never brought up, however, is the fact that any mutation will result in a loss of information due to the change in genetic material. Even in the very unusual occurrence of a so-called “beneficial” mutation, there is an ultimate loss of genetic information available to succeeding generations.Robert T. (Tommy) Mitchell, M.D., is a graduate of Vanderbilt University School of Medicine and practices Internal Medicine in Gallatin, Tennessee. He is Board Certified in Internal Medicine and is a Fellow of the American College of Physicians.
These are several of the many ways in which bacteria may develop resistance.IRQ Conflict wrote:linkFor example, bacteria may become resistant to to macrolides (erythromycin, azithromycin, clarithromycin, dirithromycin, troleandomycin, etc.) by producing a slightly altered 50S ribosomal subunit that still functions but to which the antibiotic can no longer bind (see Fig. 3A). Bacteria may become resistant to beta-lactam antibiotics (penicillins, monobactams, carbapenems, and cephalosporins) by producing altered transpeptidases (penicillin-binding proteins) with greatly reduced affinity for the binding of beta-lactam antibiotics. Bacteria may become resistant to vancomycin by producing altered cross-linking peptides in the peptidoglycan to which the antibiotic no longer bonds. Bacteria may become resistant to fluoroquinolones (norfloxacin, lomefloxacin, fleroxacin, ciprofloxacin, enoxacin, trovafloxacin, etc.) by producing altered DNA gyrase or topoisomerases