Re: Evolution and Intelligent Design
Posted: Tue Aug 14, 2012 9:00 pm
Actually the evidence provided is evidence of a system. This is what nearly every paper referenced shows. Evidence of a system. I asked this question to you before "do you know what a system is?" I also asked Do you understand what 3 dimensional spatial positioning is? Do you understand what temporal control is?Pierson5 wrote:This is exactly the issue I have with your "evidence." You are taking a small, specific aspect of biology not completely understood and claiming it as evidence for design.
This is the cool thing about evidence. We can both view it. People who are following this thread can look at the evidence for themselves. The evidence is plainly showing that there is a system of spatiotemporal positional control functioning within all life.
Then you should have no serious problems defining how it is flawed just like all the others. Right?Pierson5 wrote:The citation you gave says NOTHING about ID in the regards of this discussion. Your whole argument is just a rehashed version of Paley's watchmaker argument. Just as Behe used the flagellum (motor), you are using the same argument with 3 dimensional spatial organization. Paley's argument is flawed, Behe's argument was flawed and yours is flawed for the same reasons.
They are testing the system mostly by knockout experimentation. Determining how the positional control is being implemented and observing how cellular components are being controlled in space and time. This is an exciting field and there is some very neat new technologies that are allowing them to observe the functioning of the cell in vivo with 3 dimensional detail.Pierson5 wrote:The question I asked was specifically about the so called research you and your colleagues are currently doing. As you said above, you have presented your ideas to your colleagues and they are implementing it. Why not give us a run down of the type of experiments they are doing to test your hypothesis?
The great thing about the neat new technologies that are being developed is that it usually has a mechanical engineer operating in the biological realm that helps to make many of the instruments used in scientific study such as microscopy. Microscopes and software... systems designed by people just like me to help bring the hidden world into view. Its nice to have friends in the right places. As noted I help to research the already existing papers to look for clues that can help make determinations of where they want to explore in future scientific investigations and I also get to have a bit of fun in helping to form the 3 dimensional models of the various components that are being observed. My models are used in video's similar to this; EXPLORING THE LIVING CELL http://www.youtube.com/watch?v=ky8Xs3JhRc0
If you want names of people and specific experiments then you can look through the references I gave, there is one cited from a colleague.
That is part of it. Remember I stated quite plainly that I am an expert in the replication of 3 dimensional form. This is what I study and apply for a living and I perform research in this specific area.Pierson5 wrote:So, your "research" is looking up other people's research to look for any reference to 3D spatial control? ... EDIT: Just saw Ivellious' post
My collegues are the biology department? and just so we are clear I have a number of experimental evidences to validate the hypothesis that it requires an irreducibly complex system of spatiotemporal control of matter in order to allow for 3 dimensional replication. You simply have to look within the field of mechanical engineering to see the testing and applications of the systems.Pierson5 wrote:When I was asking if you presented your ideas to your colleagues, I meant the biology department. And just so we're clear, you don't have any type of experiments to test your hypothesis?
As for the hypothesis that such a system requires an intelligent designer the observable evidence speaks for itself there since only intelligent design has ever been observed to form such systems. So how would one test to prove that the system in life was designed? for that matter how would one test to prove that it evolved? We have a common problem. Neither of us can test the past... it is beyond the scientific method. Our only choice is to infer from current observable evidence.
You argue for natural occurance which you have never seen to form such a system and I argue for ID based on the observable evidence left behind as intelligent designers form such systems.
KBCid wrote:Replication is that simple little thing most everyone understands as the reproduction of material form. You do understand that in order to replicate material form it requires both spatial and temporal control of matter right?
Point 2 isn't true... the fact is 'You' don't understand it. I understand it quite well since the type of system you don't understand is what I make for a living and since these systems have only ever been observed as a result of ID I have every right to hypothesize that ID would be required for them to come into existence. What have you observed that forms such a system? really show me what kind of system like this you have personally observed nature forming?Pierson5 wrote:I'm not disagreeing with you that it is a requirement, and it does happen. I don't see how you go from: 1. Here is a requirement
2. We don't understand it 3. People build stuff similar 4. Therefore design
Ok let's review what I have been saying "The evidence is plainly showing that there is a system of spatiotemporal positional control functioning within all life" and I further state that "a system of spatiotemporal positional control is required for 3 dimensional replication" here is a sampling ofPierson5 wrote:I also agree with what others have said before. The citations you provide are not relevant. They do not come to the same conclusions you do. None of these papers address what our current discussion entails. One citation is discussion engineering of viable tissue and says nothing about ID in the sense you are promoting.
the evidence provide to back these statements;
Emerging paradigms of regulated microRNA processing
The exquisite spatio–temporal control of miRNA abundance is made possible, in part, by regulation of the miRNA biogenesis pathway...
...Thus, miRNAs themselves must be post-transcriptionally regulated. In fact, regulation at multiple biogenesis steps and at turnover of the mature
miRNA has now been established...
...it is clear that miRNA processing is regulated in a complex manner, and we are only beginning to understand its true nature.
http://genesdev.cshlp.org/content/24/11/1086.full
Transcriptional control of mitochondrial biogenesis: the central role of PGC-1a
Mitochondrial biogenesis
Mitochondrial proteins are encoded by the nuclear and the mitochondrial genomes. The double-strand circular mitochondrial DNA (mtDNA) is
16.5 kb in vertebrates and contains 37 genes encoding 13 subunits of the electron transport chain (ETC) complexes I,III, IV, and V, 22 transfer RNAs, and 2 ribosomal RNAs necessary for the translation.
Correct mitochondrial biogenesis relies on the spatiotemporally coordinated synthesis and import of 1000 proteins encoded by the nuclear genome, of which some are assembled with proteins encoded by mitochondrial DNA within newly synthesized phospholipid
membranes of the inner and outer mitochondrial membranes. In addition, mitochondrial DNA replication and mitochondrial fusion and ï¬ssion mechanisms must also be coordinated (Figure 1). All of these processes have to be tightly regulated in order to meet the tissue requirements.
...Mitochondrial biogenesis thus involves an intricate, complicated network of transcription factors NRFs/PPARs/ERRs that activate target genes encoding enzymes of FAO, oxidative phosphorylation, and antioxidant defences (Figure 2). PGC-1a, by co-activating, and controlling the expression of this network, directly links external physiological stimuli to the regulation of mitochondrial biogenesis
and function. Additionally, mitochondrial biogenesis involves fusion/ï¬ssion and requires protein import and processing and cardiolipin biosynthesis. http://cardiovascres.oxfordjournals.org ... 8.full.pdf
MitoGenesisDB: an expression data mining tool to explore spatio-temporal dynamics of mitochondrial biogenesis
In yeast Saccharomyces cerevisiae (S. cerevisiae), the coordinated association of more than 800 proteins (mostly encoded by the nuclear genome) are required to assemble a functional organelle (8,9). http://nar.oxfordjournals.org/content/3 ... 9.full.pdf
So in your opinion none of these references says anything like what i'm saying right?
KBCid wrote:And what does the Dover trial have to do with the irreducible complexity of the 3 dimensional spatiotemporal control system?
So you wish to misrepresent my position in an attempt to knock down the misrepresentation. the strawman argument along with the fallacy of False Analogy. You are invited to provide an argument for why it is flawed. This is where you need to be a real scientist and bring your rationale into play and define exactly how my argument is flawed. Show us all how my apple arguement is exactly like Behe's orange argument.Pierson5 wrote:As I said above, your reasoning is flawed for the exact same reasons Behe's was flawed. Every argument you have brought up was discussed and shown to be fallacious 7 years ago in the trial. "ID doesn't claim to know who the designer is," "Science is based on naturalistic philosophy and is flawed," "Flagellum (or in your case 3D spatiotemporal control systems) are evidence of design for "X" reasons." You can read the arguments in the transcripts, they are very relevant. These excerpts are from Day 1 and give a small summery of some of the things that are discussed in the trial.
Pierson5 wrote:By definition a stone archway is "irreducibly complex."
KBCid wrote:Really? So you think a stone archway is the equivalent of a 3 dimensional spatiotemporal control system? Have you ever seen a stone archway replicate itself?
Behe's mouse trap arguement is not mine nor is the stone archway. These are your strawman arguments. How about you define where my arguments are wrong.Pierson5 wrote:Behe's famous mouse trap example doesn't replicate itself. I was pointing out by definition the archway is irreducibly complex. If you take a stone away, it falls apart.
You could try.Pierson5 wrote:Couldn't I use this same argument against your "evidence" for ID? Have you ever seen something built by humans that has 3 dimensional spatiotemporal control systems and was also able to replicate itself with slight modifications?
let me introduce you to Synthia
The man-made single cell "creature", which is a modified version of one of the simplest bacteria on earth, proves that the technology works.
Now Dr Venter believes organism, nicknamed Synthia, will pave the way for more complex creatures that can transform environmental waste into clean fuel, vaccinate against disease and soak up pollution.
But his development has also triggered debate over the ethics of "playing god" and the dangers of the new technology could pose in terms of biological hazards and warfare.
"We are entering an era limited only by our imagination," he said announcing the research published in the journal Science.
"This is the first synthetic cell that's been made, and we call it synthetic because the cell is totally derived from a synthetic chromosome, made with four bottles of chemicals on a chemical synthesizer, starting with information in a computer," said Dr Venter.
http://www.telegraph.co.uk/science/7745 ... g-god.html
Observable evidence - good
KBCid wrote:The mouse trap and the flagellum which have volumes of arguments from both sides are not what I am promoting. Even if you had or have in actuality refuted those particular systems they have nothing to do with the irreducibly complex spatiotemporal control system I'm discussing. Your argument shows how little you really understand both the arguement for irreducible complexity and how little you understand about 'how' your critics believe they have rebutted it.
My cite was to define what irreducible complexity is not how it been applied.Pierson5 wrote:If these arguments are not what you are promoting, why did you provide the link!? Perhaps stop citing articles and papers that have nothing to do with what you are promoting and we won't have these misunderstandings....
KBCid wrote:You see Pearson if you had payed attention to my assertion you would have discerned the difference between Behe's proposed systems and the system that I am discussing. Behe's proposed irreducibly complex systems are supposedly debunked by the believed possibility of the evolutionary mechanism to evolve them. My system on the other hand has to occur before evolution can operate.
Let me repeat that so you don't accidently miss it "My system on the other hand has to occur before evolution can operate."I gave you this information along the way in this thread when I stated "no replication, no evolution".
I will reword this another way in case you may have a faulty understanding of what I just said. In order for irreducible complexity to even theoretically be rebutted it requires the assertion of an operating evolutionary system and that system requires some very specific things in order to operate...
Do you see the 'things' that are required for evolution to occur? I would say that alleles are a prerequisite to evolutionary operation.
I would further state that another important prerequisite is generation (replication), Definitely can't have generations without replication.
Remember what I said "no replication, no evolution" So, since an irreducibly complex 3 dimensional spatiotemporal control system is required for replication of 3 dimensional form and evolution doesn't exist until the system of replication is operational you have no imaginable mechanism to overcome the irreducible complexity point of my assertion. My system on the other hand has to occur before evolution can operate.
Pierson5 wrote:Citation? And here we have a false premise. You are assuming that evolution could not have produced this system (just as Behe assumed his flagellum could not have evolved). You are assuming that because we currently do not understand it completely, it could not have come about through evolution.
Well lets see here Pierson did you at all read this?
"Do you see the 'things' that are required for evolution to occur? I would say that alleles are a prerequisite to evolutionary operation.
I would further state that another important prerequisite is generation (replication), Definitely can't have generations without replication.
Remember what I said "no replication, no evolution" So, since an irreducibly complex 3 dimensional spatiotemporal control system is required for replication of 3 dimensional form and evolution doesn't exist until the system of replication is operational you have no imaginable mechanism to overcome the irreducible complexity point of my assertion."
You would like us to believe that evolution was functioning prior to life, prior to the existence of alleles? Prior to replication?
I am not assuming that evolution couldn't produce this system. I am stating a fact of logic here. Evolution depends on there being alleles and replication in order to function. Therefore, it did not function until both were existing. No replication, no evolution.
LoL you believe its evolution all the way down to base chemicals, no alleles needed, no replication needed... it all just evolved.
Pierson5 wrote:This may be relevant: http://www.ncbi.nlm.nih.gov/pubmed/21683595
CONCLUSIONS:
The genetic separability of spatial and temporal control modules in Caulobacter reflects their evolutionary history. DnaA is the central component of an ancient and phylogenetically widespread circuit that governs replication periodicity in Caulobacter and most other bacteria. By contrast, CtrA, which is found only in the asymmetrically dividing α-proteobacteria, was integrated later in evolution to enforce replicative asymmetry on daughter cells.
In what way is someones opinion of what may have happened relevant? Do you or they have any evidence from scientific method?
Pierson5 wrote: Either you accept paternity testing as a legitimate method of determining relatedness
Please explain to me why you accept paternity testing as a legitimate scientific method for determining relatedness, but when applied to other living organisms it is not. I'm sure you can provide plenty of "arguments" about homology.
KBCid wrote:This argument wasn't direct at me but I do have something to say about it. I will first make sure everyone understands what a paternity test involves; Paternity testing
Special locations (called loci) in human DNA display predictable inheritance patterns that could be used to determine biological relationships....
http://www.dnacenter.com/science-techno ... ience.html
Do you understand why Paternity testing uses 16 STR markers to form a legitimate method of determining relatedness? why not just use 1 STR marker?
Pierson5 wrote:The more markers used, the less likely the similarities are to be caused by chance.
hey you got something right
KBCid wrote:Legitimacy of this method of determining relatedness is not as straight forward as some assume;
http://www.bioforensics.com/articles/ch ... pion1.html
The bottom line for an assumption of relatedness is considered logically and rationally realistic when you can show enough separate genetic points (STR's) from two people that are the same.
Pierson5 wrote:The legitimacy of this method is VERY straight forward. The article you cited is warning lawyers and other scientists about individual cases that could have involved bias.
exactly. Having 16 points of reference tends to eliminate chance and bias.
Pierson5 wrote:Now, if you are claiming that 97% of scientists who accept evolution are mistaken in their evaluation of this sort of evidence, we now have a testable claim!! Feel free to re-evaluate the methods used by evolutionary/molecular/geneticists and other scientists and prove them wrong.
ok... how many genetic markers do they use to ascertain relatedness?
Pierson5 wrote:thus homology = relatedness. OR, homology =/= relatedness
KBCid wrote:Homology which describes the condition of being homologous or the similarity of position or structure is not defined by 16 separate genetic markers to provide a reasonable assumption of relatedness. Homology therefore is not the equivalent of a paternity test. It does not have the same logical or rational power that a paternity test gives based on 16 genetic markers.
Pierson5 wrote:The genetic markers are based on the differences in the DNA sequences. DNA is the bases of heredity and, I can't tell if you agree, the number of shared markers is a good measure of relatedness (accepted by courts of law and the scientific community).
There are a variety of ways to test for distinctive variations in DNA.
Yup there certainly are "ways to test for distinctive variations in DNA", Variations are one thing and relatedness is another. Now tell me how many genetic markers are used to define relatedness?
Pierson5 wrote:Comparing two people's genetic variations determines heredity. Biologists use the same process for classifying populations of organisms. This is known as phylogenetics, and wouldn't you know it, matches up perfectly with common characteristics, biogeography and the fossil record (among others, see pg 1). So, if two organisms have the same genetic variations/markers in the same location (#3 on your definition of homologous), I fail to see the false analogy there... Maybe I'm missing something?
Comparing two peoples genetic markers... 16 of them determines heredity.
Yup your missing something;
The problem posed by phylogenetics is that genetic data are only available for living taxa, and the fossil records (osteometric data) contains less data and more-ambiguous morphological characters.[6] A phylogenetic tree represents a hypothesis of the order in which evolutionary events are assumed to have occurred. http://en.wikipedia.org/wiki/Phylogenetics
KBCid wrote:We know from experience with intelligent designers that they use the same things in different ways and in different structures for various reasons so it is not beyond reason to infer that similarities found among a variety of life forms could have a common designer.
Pierson5 wrote:Intelligent designers also add novel new functions to their creations. Sometimes they create something completely new. Irrelevant. It is not just similarities. This goes back to my example using the methods of paternity testing to determine relatedness. Homology is similar structures serving similar functions. Analogous structures are those with a different origin/structure/derivation that serve a similar function. For example, the wings of bats and birds are analogous structures. The bones of the flipper of a dolphin and the hand of a human are homologous structures. Convergent evolution = evidence against evolution?
Convergent evolution = evidence for a common designer
Pierson5 wrote:In genetics, there are homologs and paralogs in gene structure. Either homology implies relatedness and paternity testing works, OR all that is wrong, in which case the experiment I provided earlier should be easy for you to conduct.
Homology is not a paternity test since it isn't based on 16 distinct genetic markers